Thursday, November 20, 2008
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SYNCOPE
Adapted from Heart Rhythm Society, Patient and Public Information Center 11/2005
Neurocardiogenic and Non-Cardiogenic Syncope
What is neurocardiogenic syncope?
- Neurocardiogenic syncope has been called the “common faint” and is also known as “vasovagal syncope”
- It can occur when a person rises to a standing position from a lying or sitting
- 25% to 40% of fainting disorders are due to neurocardiogenic syncope
Who develops neurocardiogenic syncope?
- Neurocardiogenic syncope often affects young, healthy individuals who have no history of heart disease or neurological problems
- Most common cause of fainting in young people
- Some adolescents have frequent fainting spells and may outgrow this in the mid-twenties
What happens in neurocardiogenic syncope?
- It is thought to result from a “miscommunication” between the brain and the heart and may be a result of an overactive normal reflex
- When we stand up, the brain sends signals to the blood vessels in the legs to dilate, or relax
- When the blood vessels in the lower extremities relax, blood may pool in the legs, which can reduce the amount of blood returning to the heart
- If the heart does not pump enough blood to supply the brain’s need for oxygen, lightheadedness can occur
More about what happens in neurocardiogenic syncope::
- Sometimes, the heart tries to raise the blood pressure by increasing the force of contractions and the rate of the heartbeat, which can make the problem worse
- As the heart beats stronger and faster, a “faulty” message is sent from the heart to the brain saying that the heart’s pumping chambers are filled with blood and that the blood pressure is too high
- In fact, the heart’s chambers are not full and the blood pressure is too low
- When the brain receives this “faulty” message, it slows the heartbeat even more and relaxes the blood vessels even more
- Less blood is pumped to the brain and the risk of fainting increases
How is neurocardiogenic syncope treated?
- Conservative measures to treat neurocardiogenic syncope include:
- Increase fluid intake
- Increase salt intake; avoid excess salt if blood pressure becomes elevated
- Lie down and elevate legs if an episode of lightheadedness occurs; do not attempt to “walk it off”
- Medications to treat this include:
- Beta blockers
- Salt tablets
- SSRI medications, such as paroxetine (Paxil®), sertraline (Zoloft®), fluoxetine (Prozac®) to name a few
Are there non-cardiogenic causes for syncope?
- Orthostatic syncope is the inability to maintain normal blood pressure while standing
- Due to disorders of the autonomic nervous system (ANS), which manages most of our automatic bodily functions; problems with this mechanism is called dysautonomia
- May occur in healthy people as a result of standing for long periods, exercise or overeating
- Conditions such as diabetes, Parkinson’s Disease, multiple sclerosis or conditions that cause nerve damage may contribute to this problem
- Metabolic syncope can occur in situations of hypoglycemia (low blood sugar) or hyperventilation (rapid, shallow breathing)
Non-cardiogenic syncope may be triggered by several factors:
- Any condition that causes low blood pressure
- Loss of blood volume caused by bleeding, diarrhea or vomiting
- Eating a large meal or drinking alcohol
- Vigorous exercise without re-hydration
- Medications, such as diuretics, that have led to, excessive fluid loss
- Menstruation
- “Hypersensitive” autonomic nervous system, in response to triggers, such as sneezing, coughing, urination or having a bowel movement
- Emotional or stressful situations
- Headache
- Illness or trauma
Adapted from Heart Rhythm Society, Patient and Public Information Center 11/2005
